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Michal Jazwinski, Huber R. The organizer of this program was Dr. This symposium, targeted to an audience of non-biologists, briefly described how animal models are used to elucidate aging mechanisms that might also apply to humans. It also documented the observation that manipulations that increase longevity often extend health span as well. A variety of genetic and environmental interventions that extend life span in fruit flies have been identified, and changes in gene expression patterns as a result of these interventions scale to the mean life span of the flies.
So far, more than mutations that positively influence longevity have been identified in nematodes. A variety of aging phenotypes are seen as nematodes grow older. The first life-extending single gene mutation isolated, age-1 , delays muscle wasting and nuclear deterioration, but not some other aging phenotypes such as yolk protein accumulation. The discovery in the s that dwarf mice are long-lived created a new model for aging research.
These dwarf mice are produced by mutations that either block pituitary development and therefore the production of growth hormone, or inactivate the growth hormone receptor. Both kinds of mutations reduce cancer incidence, improve retention of learning ability, and increase insulin sensitivity.
Louis, MO. This symposium elaborated in greater depth on the recent contributions that model organisms have made to our understanding of the aging process see above. A good deal of attention was devoted to the role of sirtuins in life-span regulation. An increase in Sir2 levels increases life span in fruit flies, just as it does in yeast and nematodes.
